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Samuel Gershon, MBBS, DPM; Baron Shopsin, MD; John R. Bergen, PhD; Lorraine Pinckney, MT

Arch Gen Psychiatry. 1968;19(5):611-615.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

IN 1947, Sjovall¹ reported occurrence of hemoglobin in the urine of mice following injection of serum of schizophrenics and loss of this activity of serum following heating at 56 C for one half hour. Turner and Chipps pursued this lead and in 1966 reported² that there is an antirabbit heterophil hemolysin of widespread occurrence in human serum. Their study indicated that this hemolysin was present at higher levels in the serum of schizophrenics than those of other persons.

The interest aroused by this notion of heterophil hemolysins as another³ potential "biochemical lesion" in schizophrenia prompted further investigation. Operating essentially within the same framework as Turner and Chipps, the data we collected fails to indicate a difference between schizophrenic and nonschizophrenic populations with respect to serum hemolysis of rabbit erythrocytes.

Methods

Blood was drawn in the morning from fasting subjects. One part of the . . . [Full Text PDF of this Article]

Author Affiliations
New York

From the Neuropsychopharmacology Research Unit, Department of Psychiatry, New York University School of Medicine.

Footnotes
Submitted for publication April 19, 1968.

Reprint requests to New York University Medical Center, 550 First Ave, New York 10016 (Dr. Gershon).

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